Gait disorder of subcortical arteriosclerotic encephalopathy: Binswanger's disease
Identifieur interne : 006618 ( Main/Exploration ); précédent : 006617; suivant : 006619Gait disorder of subcortical arteriosclerotic encephalopathy: Binswanger's disease
Auteurs : P. D. Thompson [Royaume-Uni] ; Marsden [Royaume-Uni]Source :
- Movement Disorders [ 0885-3185 ] ; 1987.
Descripteurs français
- Pascal (Inist)
- Wicri :
- topic : Homme.
English descriptors
- KwdEn :
- Aged, Aged, 80 and over, Ataxia (physiopathology), Ataxia (radiography), Atherosclerosis, Binswanger's disease, Binswenger subcortical encephalopathy, Cerebrovascular disease, Female, Frontal Lobe (physiopathology), Frontal Lobe (radiography), Gait, Gait disorder, Human, Humans, Intracranial Arteriosclerosis (physiopathology), Male, Middle Aged, Nervous system diseases, Neural Pathways (physiopathology), Neural Pathways (radiography), Parkinson Disease (physiopathology), Parkinson Disease (radiography), Parkinsonian‐ataxia, Syndrome, Tomography, X-Ray Computed, Videotape Recording.
- MESH :
- physiopathology : Ataxia, Frontal Lobe, Intracranial Arteriosclerosis, Neural Pathways, Parkinson Disease.
- radiography : Ataxia, Frontal Lobe, Neural Pathways, Parkinson Disease.
- Aged, Aged, 80 and over, Female, Gait, Humans, Male, Middle Aged, Syndrome, Tomography, X-Ray Computed, Videotape Recording.
Abstract
The abnormality of gait that may occur in patients with subcortical arteriosclerotic encephalopathy (Binswanger's disease) is described in 12 patients in whom difficulty walking was the presenting symptom. Their gaits had elements of both Parkinsonism and ataxia, which were most market in 4 patients unable to stand unaided. The difficulty in using their legs to walk was out of proportion to that of other movements of the lower limbs when lying or seated. In contrast, upper limb mobility and facial expression were relatively preserved. Attention is drawn to the similarity of this gait pattern to that of some patients with hydrocephalus, frontal lobe lesions, and “senile” disorders of gait. A possible mechanism for this Parkinsonian‐ataxia in these various conditions may be their propensity to damage the afferent and efferent interconnections of the leg areas of the motor and supplementary motor areas of the cerebral cortex with, the cerebellum and basal ganglia.
Url:
DOI: 10.1002/mds.870020101
Affiliations:
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Le document en format XML
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D." last="Thompson">P. D. Thompson</name><affiliation wicri:level="2"><country>Royaume-Uni</country><placeName><region type="country">Angleterre</region></placeName><wicri:cityArea>University Department of Neurology, Institute of Psychiatry and King's College Hospital Medical School, Denmark Hill, London</wicri:cityArea></affiliation></author><author><name sortKey="Marsden" sort="Marsden" uniqKey="Marsden" last="Marsden">Marsden</name><affiliation wicri:level="2"><country>Royaume-Uni</country><placeName><region type="country">Angleterre</region></placeName><wicri:cityArea>University Department of Neurology, Institute of Psychiatry and King's College Hospital Medical School, Denmark Hill, London</wicri:cityArea></affiliation></author></analytic><monogr></monogr><series><title level="j">Movement Disorders</title><title level="j" type="sub">Official Journal of the Movement Disorder Society</title><title level="j" type="abbrev">Mov. Disord.</title><idno type="ISSN">0885-3185</idno><idno type="eISSN">1531-8257</idno><imprint><publisher>Wiley Subscription Services, Inc., A Wiley Company</publisher><pubPlace>Hoboken</pubPlace><date type="published" when="1987">1987</date><biblScope unit="vol">2</biblScope><biblScope unit="issue">1</biblScope><biblScope unit="page" from="1">1</biblScope><biblScope unit="page" to="8">8</biblScope></imprint><idno type="ISSN">0885-3185</idno></series><idno type="istex">104B7C44CB20F2A7C21FDB67F28579D18571F2A9</idno><idno type="DOI">10.1002/mds.870020101</idno><idno type="ArticleID">MDS870020101</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0885-3185</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Aged</term><term>Aged, 80 and over</term><term>Ataxia (physiopathology)</term><term>Ataxia (radiography)</term><term>Atherosclerosis</term><term>Binswanger's disease</term><term>Binswenger subcortical encephalopathy</term><term>Cerebrovascular disease</term><term>Female</term><term>Frontal Lobe (physiopathology)</term><term>Frontal Lobe (radiography)</term><term>Gait</term><term>Gait disorder</term><term>Human</term><term>Humans</term><term>Intracranial Arteriosclerosis (physiopathology)</term><term>Male</term><term>Middle Aged</term><term>Nervous system diseases</term><term>Neural Pathways (physiopathology)</term><term>Neural Pathways (radiography)</term><term>Parkinson Disease (physiopathology)</term><term>Parkinson Disease (radiography)</term><term>Parkinsonian‐ataxia</term><term>Syndrome</term><term>Tomography, X-Ray Computed</term><term>Videotape Recording</term></keywords><keywords scheme="MESH" qualifier="physiopathology" xml:lang="en"><term>Ataxia</term><term>Frontal Lobe</term><term>Intracranial Arteriosclerosis</term><term>Neural Pathways</term><term>Parkinson Disease</term></keywords><keywords scheme="MESH" qualifier="radiography" xml:lang="en"><term>Ataxia</term><term>Frontal Lobe</term><term>Neural Pathways</term><term>Parkinson Disease</term></keywords><keywords scheme="MESH" xml:lang="en"><term>Aged</term><term>Aged, 80 and over</term><term>Female</term><term>Gait</term><term>Humans</term><term>Male</term><term>Middle Aged</term><term>Syndrome</term><term>Tomography, X-Ray Computed</term><term>Videotape Recording</term></keywords><keywords scheme="Pascal" xml:lang="fr"><term>Athérosclérose</term><term>Encéphalopathie souscorticale Binswenger</term><term>Homme</term><term>Système nerveux pathologie</term><term>Trouble marche</term><term>Vaisseau sanguin encéphale pathologie</term></keywords><keywords scheme="Wicri" type="topic" xml:lang="fr"><term>Homme</term></keywords></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">The abnormality of gait that may occur in patients with subcortical arteriosclerotic encephalopathy (Binswanger's disease) is described in 12 patients in whom difficulty walking was the presenting symptom. Their gaits had elements of both Parkinsonism and ataxia, which were most market in 4 patients unable to stand unaided. The difficulty in using their legs to walk was out of proportion to that of other movements of the lower limbs when lying or seated. In contrast, upper limb mobility and facial expression were relatively preserved. Attention is drawn to the similarity of this gait pattern to that of some patients with hydrocephalus, frontal lobe lesions, and “senile” disorders of gait. A possible mechanism for this Parkinsonian‐ataxia in these various conditions may be their propensity to damage the afferent and efferent interconnections of the leg areas of the motor and supplementary motor areas of the cerebral cortex with, the cerebellum and basal ganglia.</div></front></TEI><affiliations><list><country><li>Royaume-Uni</li></country><region><li>Angleterre</li></region></list><tree><country name="Royaume-Uni"><region name="Angleterre"><name sortKey="Thompson, P D" sort="Thompson, P D" uniqKey="Thompson P" first="P. D." last="Thompson">P. D. Thompson</name></region><name sortKey="Marsden" sort="Marsden" uniqKey="Marsden" last="Marsden">Marsden</name></country></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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